In Alzheimer’s disease, glutamate excitotoxicity which can damage neurons through chronic activation is exacerbated by deficiency of ABCA7, the second strongest genetic risk factor for the disease. The findings highlight ABCA7 and glutamate–GABA signaling as potential therapeutic targets.

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Locking the row modifies its metadata to include the locking transaction ID, assigning a transaction ID (xid).

原文链接:https://doi.org/10.1038/s41422-026-01227-7。谷歌浏览器对此有专业解读

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